Professional Documents
Culture Documents
F. HABITS/HABITUATION
1. Smoking
2. Regular alcohol intake
3. Drug abuse/use
-FHT
- AOG
- position, presentation
- abnormalities
3. Detects placental location or placental abnormality
4. An important aid in high-risk procedures like amniocentesis
Preparation:
1. Advices mother to drink 1 quarter of water 2 hrs before procedure.
2.Instruct not to void (Ultrasound)
In amniocentesis w/ ultrasound to offer visualization, the mother should void to prevent injuring the distended bladder w/ needle insertion.
3. Transmission gel is spread over maternal abdomen
4. Psychological support is given to the mother/father
-confinement is not needed
-no need for dye and there is no x-ray radiation
-takes about 30 min. to accomplish
2. NON-STRESS TEST (NST)
- Observation of FHR related to movement
- Test of fetal well-being
Preparation:
1. Position semi-fowlers or left lateral
2. Check BP
3. Explain :
- Procedure takes : 30-60 min.
- Mother needs to activate mark button w/ each fetal movement
- Does not need hospitalization ambulatory basis
4. Requires external electronic monitoring of FHT w/ ultrasound transducer and tocodynamometer to trace fetal activity and or uterine activity
Interpretation:
1. Normal : Reactive
- Increased FHR (acceleration)
- 15 bpm above baseline
- lasting 15 sec or more in a 10-20 min period w/ fetal movement
2. Abnormal : Non reactive
-No FHR acceleration w/ fetal movement
Implication of Results:
1. Normal : High risk pregnancy continues
2. Abnormal : Mother needs another test, may be BPP
3. OXYTOCIN CHALLENGE TEST (OCT) OR CONTRACTION STRESS TEST (CST)
Purposes :
1. Observation of response of the fetus to individual uterine contractions
Implications:
1. Decreased L/S ratio
2. Fetal blood false high levels of AFP
Aftercare:
1. Monitor 30-60 min
2. Observe for side effects such as :
-vaginal discharge
-increase uterine/fetal activity
- fever and chills
Analysis of amniotic fluid
1. TO determine fetal lung maturity
L/S ratio: 2:1 means mature lungs
2. Determination of AOG
-Creatinine levels : 2.0 mg 36 wks AOG
-Nile blue stain (lipid cells) when 20 % of cells are stained w/ orange means fetal weight is at least 2500 g
3. Alpha-fetoprotein levels :
High levels : presence of neural defects such as :
-aspina bifida
-tracheoesophageal atresia
4. Genetic disorder
5. Rh incompatibility : Increased levels of bilirubin identified isoimmunization; evaluated for intrauterine transfusion or deliver
6. Inborn errors metabolism : biochemical analysis of fetal cell enzymes
*Rh- rhesus
7. Fetal distress: passage of meconium in cephalic presentation (not significant in breech presentation)
8. Sex-linked disorders : sex chromosome determination
7. X-RAY LATERAL PELVIMETRY
-Indications for radiography to determine pelvic size and shape
- suspected cephalopelvic disproportion
-history of injury/disease of pelvis and spine
-previous difficult delivery
- cases of maternal deformity or limp
8. SERIAL ESTRIOL DETERMINATION
-Measures feto-placental wellbeing
-specimens: serum/ 24 hr urine
Results:
1. Normal : gradual increase in serial estriol w/c is 12-50 mg/day at term
2. Abnormal : sudden drop of less than 50% of level means fetal distress
3. Persistent low levels means fetal wellbeing is compromised
9. CHORIONIC VILLI SAMPLING (CVS)
- Earliest test possible on fetal cells
- Sample obtained by slender catheter passed through cervix to implantation site
10. PUBS
- Used in 2nd and 3rd tri.
-Uses ultrasound to locate umbilical cord
-Cord blood aspirated and tested
CARDIAC DISEASE
- Caused by inability to cope w/ added volume and increase cardiac output that occurs during pregnancy.
Class I- Uncompromised
- No symptoms of insufficiency
- No limits on physical activity
*Poor prognosis for vaginal delivery; no lithotomy and valsalva maneuver w/c can trigger cardiac arrest ; no rectal suppository w/c can trigger valsalva maneuver
CONGENITAL ANOMALIES
- Atrial septal disease
- Ventricular septal defect
- Occur from congenital or rheumatic heart disease
- Pulmonary stenosis
- Coarction of Aorta
1. RHEUMATIC HEART DISEASE (rheumatic endocarditis)
- Endocarditis w/ scar tissue formation on the mitral, aortic, tricuspid valves w/ resulting stenosis or insufficiency
- Patient w/ mitral valve prolapsed may require prophylactic therapy during labor
Pathophysio :
valve stenosis Decrease blood flow , work on the heart chambers increase regurgitation occurs through incomplete closed valve , workload on heart increases
Assessment
1. Tachycardia
2. Dyspnea
3. Diastolic murmur
4. Crackles at lung bases
5. Hemoptysis
6. Orthopnea
Management
1. Activity limitation
2. Close medical supervision
3. Rest
4. Limited sodium intake
5. Prophylactic antibiotics
6. Serial ultrasound, non stress test, and BPP
Nursing intervention
1. Assess maternal V/S and cardiopulmonary status
2. Monitor weight gain
3. Prescribed medication
4. Frequent prenatal visit
5. Report danger signs and symptoms
6. Assess nutritional pattern
7. Frequent rest
8. Report any signs and symptoms of infection
9. Advise rest in lateral recumbent position
10. Anticipate use of epidural anesthesia labor
11. Close observation for changes during labor
12. Monitor V/S
13. W/ severe heart failure used anticoagulant and cardiac glycoside therapy
14. Encourage ambulation
15. Administer of a prophylactic antibiotic
16. Instruct to avoid high altitude, smoking area, crowded areas, drinking alcohol, and food w/ caffeine and smoking is prohibited
Remember
*should avoid
- infection
- excessive weight
- edema
- anemia
2. DIABETES MELLITUS
- Metabolic disorder characterized by hyperglycemia resulting from lack of insulin
- Endocrine disorder of carbohydrate, proteins and fat metabolism
Gestational DM (GDM)
Pregestational DM (PGDM) prior to pregnancy
Classification:
1. Type I (IDDM) Insulin dependent DM
2. Type II (NIDDM) Non insulin dependent DM
Factors :
1. Hereditary
2. Environment
3. Lifestyle
Susceptible for type I:
1. Production of autoantibodies against beta cells of pancreas
2. Restriction of beta cells
3. Decline or lack of insulin
4. Hyperglycemia
5. Lipolysis
6. Protein catabolism
Assessment:
1. Hyperglycemia
2. Glycosuria
3. Polyuria
4. Polydipsia
5. Weight loss
6. Increased incidence of candidal infections
7. Hydramnios
8. S/S of macrovascular and microvascular changes
9. Peripheral vascular disease
10, Retinopathy
11. Nephropathy
12. Neuropathy
Diagnostic test
1. Screening w/ an oral glucose challenge test
2. 100 g glucose load used at 24-28 wks gestation
Management
1. Blood glucose monitoring
2. Target glucose level of FBS
3. Diet, exercise and insulin administration
Type 1
1. Monitoring of glucose level
2. Evaluation of glycosylated hgb (HbA1c) level every 3-4 mos.
Interventions
1. Monitor patient status throughout the pregnancy
2. Review result of fingerstick blood glucose monitoring
3. Follow up lab results
4. Encourage consistent exercise program
5. Diabetic care management
6. Assist w/ preparation for labor
7. Assess possible complications and effects
Mother:
1. Has 30% to 40% chance of developing DM in 1-25 yrs.
2. Dystocia because of large infant
3. Increased risk for postpartum hemorrhage due to over distention of uterus
4. Polyhydramnios
5. Gestational hypertension
6. Maternal mortality
7. Retinopathy and nephropathy
8. Preterm delivery
9. UTI
IV. Narcotics
- heroin, methadone, morphine
Maternal effects:
1. Spontaneous abortion
2. ROM
3. Preterm labor
4. Increase incidence of STD
5. HIV exposure
6. Hepatitis
7. Malnutrition
Fetal effects
1. IUGR
2. Perinatal asphyxia
3. Intellectual impairment
3. Neonatal abstinence syndrome
4. Neonatal infections
5. Neonatal death (SIDS, child abuse and neglect)
V. Cocaine
- cracked
- Causes decrease in blood flow to the heart
- Platelet aggregation
- Brain (seizures)
- Intestines
- Uterus
-Increase in fetal neurotransmitter causes teratogenic effects
Maternal effects:
1. Hyperarousal state
2. Generalized vasoconstriction
3. HPN
4. Spontaneous abortion
5. Abruptio placenta
6. Preterm labor
7. Cardiovascular complications
8. Seizure
9. STD
Fetal effects:
1. Stillbirth
2. Poor feeding reflexes
3. N/V
4. Diarrhea
5. Decrease in intellectual development
6. Prune-belly syndrome resulting from absence of abdominal muscle
VI. Amphetamine
- speed in ice
- When processed in crystals to smoke methampethetamines ecstasy
Maternal effects:
1. Malnutrition
2. Tachycardia
3. Withdrawal symptoms
Fetal effects:
1. Increase in risk for Cardiac anomalies
2. Cleft palate
3. IUGR
4. Withdrawal syndrome
5. Fetal death
VII. Marijuana
- pot , grass
- Delta-9-tetrahydrocannabiral crosses placenta
- Takes 30 days to be excreted
- Increase in carbon monoxide 5x more than tobacco
- Detrimental when used during 1st tri. where fetal organs are being formed
Maternal effects:
1. High incidence of anemia
2. Inadequate weight gain
Fetal effects:
1. IUGR
2. Neonatal tremors
3. Sensitivity to light
Assessment:
1. Malnutrition
2. STD
3. poor self image