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A number of different
sensations experienced by patients are probably included in this category. Dyspnea is the
most common cause of respiratory limitation of activity in patients with pulmonary disease.
The sensations produced in normal people by the above experiments are probably not the
same, even though they frequently are lumped into the single category of dyspnea. Similarly,
it is probable that patients with complaints of dyspnea do not all have identical sensations.
Dyspnea is a common complaint in patients with obstructive and restrictive pulmonary
disorders. In addition, patients with chest wall abnormalities such as kyphoscoliosis and
patients with neuromuscular disease frequently complain of dyspnea. What the mechanisms
for these various dyspneic sensations are is not completely clear.
Mechanism of dyspnea
In this learning issue we talk about progressive(worsening) dyspnea. What does progressive
dyspnea mean? By progressive here it means the difficulty and uncomfortness of breathing is
worsening gradually. If remain untreated, it may lead to breathing failure and eventually
death. The progress may take several days, weeks, months or years, also it may happen in a
emergency case which only last a few hours and need immadiate treatment. Below we will
list some disease and abnormalities which involve progressive dyspnea.
Type of dyspnea
Dyspnea can be symptomatic of a variety of disorders, both acute and chronic. Acute
conditions include acute infections and inflammations of the respiratory tract, obstruction by
an inhaled foreign object, anaphylactic swelling of the tracheal and bronchial mucosa, and
traumatic injury to the chest. Chronic disorders usually fall into the category of Chronic
Airflow Limitation, or are associated with pulmonary edema and congestive heart failure. A
fat embolism resulting from the release of fat particles from bone marrow at the time of a
fracture of a long bone also can cause dyspnea
Acute shortness of breath, which comes on suddenly, over the course of minutes or hours,
usually has different causes than chronic shortness of breath, which develops over weeks or
months. The most common causes of acute shortness of breath involving worsening dyspnea
include:
Chronic shortness of breath — Some of the same things that cause acute shortness of breath
can also cause chronic shortness of breath. For example, asthma symptoms can come and go
over months or years. Heart failure can also continue to cause shortness of breath over
months or years.The most common causes of chronic shortness of breath include:
Asthma
Chronic Obstructive Pulmonary Disease
A few of the most common disorders are outlined here. Patients with chronic
obstructive pulmonary disease (COPD), which comprises emphysema and chronic
bronchitis, generally present with a long history of cigarette smoking and gradually
progressive dyspnea over a number of years. Pulmonary function abnormalities
frequently progress for years before clinical dyspnea occurs. Most patients do not
have day-to-day variation in their symptoms, but they may have exacerbations during
the winter months. A productive cough is a frequent associated symptom, and the
sputum may become purulent during exacerbations.
Interstitial lung disease, a collection of lung disorders that involve damage or
scarring of lung tissue. Patients with pulmonary fibrosis generally present with
progressive and relentless dyspnea with a variable time course. Frequently the only
associated symptom is a non-productive cough. Extrapulmonary manifestations of
diseases associated with pulmonary fibrosis may be present. The occupational and
environmental history is particularly important.
Mahler DA, O'Donnell DE. 2014. Dyspnea: Mechanisms, Measurement, and Management.
3rd Edition. NW: CRC Press. p. 3.
Miller-Keane Encyclopedia and Dictionary of Medicine, Nursing, and Allied Health, Seventh
Edition. 2003. Saunders, Elsevier, Inc.
Bass JB JR. 1990. Dyspnea. In Walker HK, Hall WD, Hurst JW (Eds.). Clinical Methods:
The History, Physical, and Laboratory Examinations. 3rd edition. Boston: Butterworths.
Chapter 36. Available from: http://www.ncbi.nlm.nih.gov/books/NBK357/